Includes bibliographical references and index.
|Statement||edited by Jesus A. Bianco.|
|Contributions||Bianco, Jesus A.|
|LC Classifications||RC692 .S77 2006|
|The Physical Object|
|Pagination||xi, 156 p. :|
|Number of Pages||156|
Simon et al. used published data of prospective studies of subclinical disease to calculate the incidence of cardiovascular end-points associated with subclinical atherosclerosis as identified by carotid IMT, carotid ultrasound, elevated coronary artery calcium on CT examination, decreased ankle–arm index pressure assessed by Doppler, or pulse wave velocity assessed by mecanography. They found that while the yearly incidence of coronary events was atherosclerosis Cited by: Fernandes VR, Polak JF, Edvardsen T, et al. Subclinical atherosclerosis and incipient regional myocardial dysfunction in asymptomatic individuals: the Multi-Ethnic Study of Atherosclerosis (MESA). J Am Coll Cardiol ; Lakshmanan SB, M.J. Something old predicting something new. Circ Cardiovasc Imaging ;12;e This coronary sign of subclinical atherosclerosis may be used in the future to preselect those who would benefit from pharmaceutic intervention and avoid treatment in others. Meanwhile, McEvoy et al. (Circulation (2)–, ) have proposed CAC as a guide for a personalized risk-based approach to initiation and intensification of Author: Raimund Erbel, Nils Lehmann, Andreas Stang, Sofia Churzidse, Susanne Moebus, Karl-Heinz Jöckel. Intima-media thickness is a marker of subclinical atherosclerosis at the level of the carotid arteries. It is measured by high-frequency (≥ 8 MHz) ultrasound transducers in both carotid arteries, on the distal straight 1 cm off the common carotid arteries, the carotid bifurcations and the proximal artery and 1 cm of the internal carotid arteries.
The aims of this study were to: (a) investigate association of sICAM-1, sVCAM-1, omentin-1 and other non-traditional risk factors with subclinical atherosclerosis; (b) examine the diagnostic value. They do thus have a subclinical atherosclerotic disease. We have developed a risk assessment tool based on the conventional risk factors to address subclinical atherosclerosis. This tool was derived from our population-based study of around men and women (35 – 70 years of age) examined from to applying carotid ultrasound to. As the process of atherosclerosis works its damage on the artery lining it becomes thicker and plaque begins to develop. High frequency ultrasound is an excellent tool for identifying these early, subclinical stages of atherosclerosis because it is painless, harmless, inexpensive, and quick. Identifying Higher Risk Patients. Subclinical atherosclerosis—identified with vascular ultrasound and coronary artery calcium (CAC) screening using CT—was present in % of participants. Disease was present in the carotid arteries (%), infrarenal aorta (%), and iliofemoral arteries (%).
The text focuses on accurate, yet underutilized, measures of subclinical atherosclerosis, notably coronary artery calcium scanning and carotid intima-media thickness measurement. The authors introduce a comprehensive approach to identifying the vulnerable patients (asymptomatic individuals at risk of a near future adverse event). The text focuses on accurate, yet underutilized, measures of subclinical atherosclerosis, notably coronary artery calcium scanning and carotid intima-media thickness measurement. The authors introduce a comprehensive approach to identifying the vulnerable patients (asymptomatic individuals at risk of a near future adverse event). PESA-CNIC (Progression of Early Subclinical Atherosclerosis–Centro Nacional de Investigaciones Cardiovasculares)-Santander is an observational prospective cohort study that recruited 4, male and female employees of Santander Bank in Madrid from 40 to 54 years of age. All participants were free of known cardiovascular disease (CVD). Systemic lupus erythematosus (SLE) can affect various systems and organs. The most severe forms of the disease affect the kidneys, the central nervous system, and the heart. Cardiac and cardiovascular system diseases are inter alia caused by atherosclerosis, vasculitis, and thromboembolic events. Patients with SLE are at a higher risk of developing accelerated atherosclerosis.